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Alcohol and Alcoholism Vol. 35, No. 2, pp. 210-211, 2000
© 2000 Medical Council on Alcoholism

Letters to the Editors

ALCOHOL-DEPENDENT PATIENTS WITH NEUROENDOCRINE EVIDENCE FOR REDUCED DOPAMINE D2 RECEPTOR FUNCTION HAVE DECREASED PLATELET MONOAMINE OXIDASE-B ACTIVITY

Ulf Berggren, Claudia Fahlke1 and Jan Balldin*

Institute of Clinical Neuroscience, Department of Psychiatry and Neurochemistry, Göteborg University, Sahlgrenska University Hospital/Mölndal, SE-431 80 Mölndal and
1 Department of Psychology, Göteborg University, Box 500, SE-405 30 Göteborg, Sweden

Received 11 June 1999; first review notified 9 August 1999; accepted 16 August 1999

This letter is a report of a re-evaluation of the results obtained in an earlier study of ours (Balldin et al., 1994Go) of platelet monoamine oxidase (MAO)-B activity in alcoholics with reduced dopamine (DA) D2 receptor function, as assessed by the growth hormone (GH) response to the D1/D2 agonist apomorphine (APO), which was published in this journal. The reason for the re-evaluation of the results in our study is a Letter to the Editors of this journal by Farren and Dinan (1996) reporting on platelet MAO-B activity in alcoholics with reduced DA D2 receptor function, as assessed by the GH response to the DA D2 receptor agonist bromocriptine. In the report by Farren and Dinan (1996), four of eight alcoholics had no increase in GH above baseline, whereas all eight subjects in the control group had the expected GH . . . [Full Text of this Article]

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