Skip Navigation


Alcohol and Alcoholism Advance Access originally published online on November 18, 2004
Alcohol and Alcoholism 2005 40(2):155-156; doi:10.1093/alcalc/agh106
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow All Versions of this Article:
40/2/155    most recent
agh106v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (6)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by AGABIO, R.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by AGABIO, R.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?


Alcohol & Alcoholism Vol. 40, No. 2 © Medical Council on Alcohol 2005; all rights reserved

LETTER TO THE EDITOR

THIAMINE ADMINISTRATION IN ALCOHOL-DEPENDENT PATIENTS

ROBERTA AGABIO

Bernard B. Brodie Department of Neuroscience, University of Cagliari, Viale Diaz, 182, 1-09126 Cagliari, Italy

(Received 28 August 2004; first review notified 12 September 2004; in revised form 28 September 2004; accepted 7 October 2004)

The first 10% of the full text of this article appears below.

Thiamine (vitamin B1) is a water-soluble vitamin that is involved in the metabolism of glucose and lipids as well as in the production of glucose-derived neurotransmitters (see Cook et al., 1998Go). Its deficiency leads to a variety of neurological and cardiovascular symptoms and signs. Early symptoms may include fatigue, weakness and emotional disturbance, whereas prolonged gradual deficiency may lead to a form of polyneuritis (known as dry beriberi), cardiac failure or peripheral oedema (wet beriberi) (Thomson, 2000Go).

Severe thiamine deficiency (TD) may result in the . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Alcohol AlcoholHome page
A. D. THOMSON and E. J. MARSHALL
THE TREATMENT OF PATIENTS AT RISK OF DEVELOPING WERNICKE'S ENCEPHALOPATHY IN THE COMMUNITY
Alcohol Alcohol., March 1, 2006; 41(2): 159 - 167.
[Abstract] [Full Text] [PDF]